It is time to learn genetics to better understand your body and symptoms you may experience.
For over 15 years I treated hundreds of patients and use for their holistic diagnosis and treatment a Mars III, a complex digital radionics device. With Mars III I can easily check the chromosomes and genes of my patients.
For all my over 500 patients their health condition is always determined by their genetics and/or by the overload with bacteria, parasites and toxins. If these are the real causes of any health condition it is obviously that only by correcting the microbiology and re-informing our genetic code you can trigger a real healing process, everything else is just symptoms sedation with many side effects. Whyle many doctors are seriously paying attention to detoxification, elimination of parasites, clearing of bacterial and viral overload, almost no physician or naturopathic doctor pay attention to the genetics of their patients. Why not? Because they simply do not have the necessary tools and evtl. the knowledge to do ´genetic diagnosis and treatments. Well our genetic code is nothing else as a code of information to guide all regulative and metabolic processes taking place in our over 50 trillions of cells. So if the information received are wrong the cells will mutate, degenerate or die. How to reinform our genetic code? It is similar to a bug in our computer software.
Reinforming the system, i.e. clearing the bug, will give our body computer a new chance to gain back its “good software”. If all this is already known why not using that technology which deals only with bio-information, RADIONICS. With a reliable and very efficient, digital radionic device like Mars III (digital because of the huge number of data needed to be processed) you can gently reinform the genetics of your patients and thus to really cure their health conditions. If a genetic assessment with a good laboratory will cost you over $100000 and if a genetic treatment goes even closer to a million of dollars, try to do a much better genetic job by using a device which costs less than $ 15000. This is not a sales pitch, it is an invitation to try something new and to give your patients an excellent prevention and efficient treatments. Therefore I am inviting you to contact me and have a life demo of what I can do with Mars III and to talk to my patients who were or are in treatment with our team.
What is chromosome 7?
Humans normally have 46 chromosomes in each cell, divided into 23 pairs. Two copies of chromosome 7, one copy inherited from each parent, form one of the pairs. Chromosome 7 spans about 159 million DNA building blocks (base pairs) and represents more than 5 percent of the total DNA in cells.
Identifying genes on each chromosome is an active area of genetic research. Because researchers use different approaches to predict the number of genes on each chromosome, the estimated number of genes varies. Chromosome 7 likely contains 900 to 1,000 genes that provide instructions for making proteins. These proteins perform a variety of different roles in the body.
Genes on chromosome 7 are among the estimated 20,000 to 25,000 total genes in the human genome.
How are changes in chromosome 7 related to health conditions?
Many genetic conditions are related to changes in particular genes on chromosome 7. This list of disorders associated with genes on chromosome 7 provides links to additional information.
Changes in the structure or number of copies of a chromosome can also cause problems with health and development. The following chromosomal conditions are associated with such changes in chromosome 7.
- Changes in the number or structure of chromosome 7 occur frequently in human cancers. These changes are typically somatic, which means they are acquired during a person’s lifetime and are present only in tumor cells. Many forms of cancer are associated with damage to chromosome 7. In particular, changes in this chromosome have been identified in cancers of blood-forming tissue (leukemias) and cancers of immune system cells (lymphomas). A loss of part or all of one copy of chromosome 7 is common in myelodysplastic syndrome, which is a disease of the blood and bone marrow. People with this disorder have an increased risk of developing leukemia. Studies suggest that some genes on chromosome 7 may play critical roles in controlling the growth and division of cells. Without these genes, cells could grow and divide too quickly or in an uncontrolled way, resulting in a cancerous tumor. Researchers are working to identify the genes on chromosome 7 that are involved in the development and progression of cancer.
- Greig cephalopolysyndactyly syndrome
- Abnormalities of chromosome 7 are responsible for some cases of Greig cephalopolysyndactyly syndrome. These chromosomal changes involve a region of the short (p) arm of chromosome 7 that contains the GLI3 gene. This gene plays an important role in the development of many tissues and organs before birth. In some cases, Greig cephalopolysyndactyly syndrome results from a rearrangement (translocation) of genetic material between chromosome 7 and another chromosome. Other cases are caused by the deletion of several genes, including GLI3, from the short arm of chromosome 7. The loss of multiple genes can cause a more severe form of this disorder called Greig cephalopolysyndactyly contiguous gene deletion syndrome. People with this form of the disorder have characteristic developmental problems involving the limbs, head, and face along with seizures, developmental delay, and intellectual disability.
- Russell-Silver syndrome
- People normally inherit one copy of each chromosome from their mother and one copy from their father. For most genes, both copies are expressed, or “turned on,” in cells. For some genes, however, only the copy inherited from a person’s father (the paternal copy) is expressed. For other genes, only the copy inherited from a person’s mother (the maternal copy) is expressed. These parent-specific differences in gene expression are caused by a phenomenon called genomic imprinting. Chromosome 7 contains a group of genes that normally undergo genomic imprinting. Abnormalities involving these genes appear to be responsible for many cases of Russell-Silver syndrome. In 7 percent to 10 percent of cases of Russell-Silver syndrome, people inherit both copies of chromosome 7 from their mother instead of one copy from each parent. This phenomenon is called maternal uniparental disomy (UPD). Maternal UPD causes people to have two active copies of maternally expressed imprinted genes rather than one active copy from the mother and one inactive copy from the father. These individuals do not have a paternal copy of chromosome 7 and therefore do not have any copies of genes that are active only on the paternal copy. In cases of Russell-Silver syndrome caused by maternal UPD, an imbalance in active paternal and maternal genes on chromosome 7 underlies the signs and symptoms of the disorder.
- Saethre-Chotzen syndrome
- Some cases of Saethre-Chotzen syndrome result from abnormalities of chromosome 7. These chromosomal changes involve a region of the short (p) arm of chromosome 7 that contains theTWIST1 gene. This gene plays an important role in early development of the head, face, and limbs. The chromosome abnormalities responsible for Saethre-Chotzen syndrome include translocations of genetic material between chromosome 7 and another chromosome, a rearrangement of genetic material within chromosome 7 (an inversion), or the formation of an abnormal circular structure called a ring chromosome 7. Each of these chromosomal changes alters or deletes the TWIST1 gene and may also affect nearby genes. When Saethre-Chotzen syndrome is caused by a chromosomal deletion instead of a mutation within the TWIST1 gene, affected children are much more likely to have intellectual disability, developmental delay, and learning difficulties. These features are typically not seen in classic cases of Saethre-Chotzen syndrome. Researchers believe that a loss of other genes on the short arm of chromosome 7 may be responsible for these additional features.
- Williams syndrome
- Williams syndrome is caused by the deletion of genetic material from a portion of the long (q) arm of chromosome 7. The deleted region, which is located at position 11.23 (written as 7q11.23), is designated the Williams-Beuren region. This region includes more than 25 genes, and researchers believe that the characteristic features of Williams syndrome are probably related to the loss of several of these genes. While a few of the specific genes related to Williams syndrome have been identified, the relationship between most of the genes in the deleted region and the signs and symptoms of Williams syndrome is unknown. Other chromosomal conditions Whereas Williams syndrome is caused by a deletion of genes in the Williams-Beuren region of chromosome 7, another syndrome is caused by the abnormal duplication (copying) of genes in this region. This duplication appears to be associated with delayed expressive language skills (vocabulary and the production of speech) and delayed development. Very few people with a duplication of the Williams-Beuren region have been identified.
Other changes in the number or structure of chromosome 7 can cause delayed growth and development, intellectual disability, distinctive facial features, skeletal abnormalities, delayed speech, and other medical problems. Changes in chromosome 7 include an extra copy of some genetic material from this chromosome in each cell (partial trisomy 7) or a missing segment of the chromosome in each cell (partial monosomy 7). In some cases, several DNA building blocks (nucleotides) are abnormally deleted or duplicated in part of chromosome 7. A circular structure called ring chromosome 7 is also possible. Ring chromosomes occur when a chromosome breaks in two places and the ends of the chromosome arms fuse together to form a circular structure.
Is there a standard way to diagram chromosome 7?
Geneticists use diagrams called ideograms as a standard representation for chromosomes. Ideograms show a chromosome’s relative size and its banding pattern. A banding pattern is the characteristic pattern of dark and light bands that appears when a chromosome is stained with a chemical solution and then viewed under a microscope. These bands are used to describe the location of genes on each chromosome.
Where can I find additional information about chromosome 7?
You may find the following resources about chromosome 7 helpful. These materials are written for the general public.
- Additional NIH Resources – National Institutes of Health (3 links)
- Educational resources – Information pages (3 links)
- MedlinePlus – Health information
You may also be interested in these resources, which are designed for genetics professionals and researchers.
Where can I find general information about chromosomes?
The Handbook provides basic information about genetics in clear language.
- What is DNA?
- What is a chromosome?
- How many chromosomes do people have?
- Can changes in chromosomes affect health and development?
These links provide additional genetics resources that may be useful.
What glossary definitions help with understanding chromosome 7?
bone marrow ; cancer ; cell ; chromosome ; contiguous ; contiguous gene deletion syndrome ;critical region ; deletion ; developmental delay ; DNA ; duplication ; expressed ; gene ; gene deletion ;gene expression ; immune system ; imprinting ; inversion ; leukemia ; maternal ; monosomy ; mutation ;myelodysplastic syndrome ; progression ; rearrangement ; ring chromosomes ; syndrome ; tissue ;translocation ; trisomy ; tumor ; uniparental disomy
You may find definitions for these and many other terms in the Genetics Home Reference Glossary.
See also Understanding Medical Terminology.
The resources on this site should not be used as a substitute for professional medical care or advice. Users seeking information about a personal genetic disease, syndrome, or condition should consult with a qualified healthcare professional. See How can I find a genetics professional in my area? in the Handbook.